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Breakthrough brain sugar discovery turns the tables on Alzheimer's disease | Sugar stores in the brain were previously thought to be unimportant
Remarkable new findings about the sugar stores in neurons have unlocked an entire new method of treating Alzheimer's disease and other cognitive decline, and it goes a long way to explaining why there's a growing body of evidence linking GLP-1 weight loss drugs to protection from dementia.
Remarkable new findings about the sugar stores in neurons have unlocked an entire new method of treating Alzheimer's disease and other cognitive decline, and it goes a long way to explaining why there's a growing body of evidence linking GLP-1 weight loss drugs to protection from dementia. “By increasing GlyP activity, the brain cells could better detoxify harmful reactive oxygen species, thereby reducing damage and even extending the lifespan of tauopathy model flies,” said study lead Sudipta Bar. This mechanism is made more efficient by higher levels of GlyP, which the researchers were able to demonstrate in human neurons sourced from frontotemporal dementia(FTD) patients, and shows that this treatment could benefit those with neurodegenerative conditions by kickstarting the cleaning process that had otherwise come to a halt.
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