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Following a study in mice, scientists have now confirmed that silencing the MTCH2 protein in muscle tissue leads to energy-deprived human cells seeking out fat for fuel, while blocking the body's ability to store extra fat cells.
Following on from a remarkable study in mice, scientists have now confirmed that silencing a certain protein in muscle tissue leads to energy-deprived human cells seeking out fat for fuel, while blocking the body's ability to store extra fat cells.
The team has found that silencing MTCH2 protein expression in muscle tissue, which was first demonstrated in mice, also made human cells essentially "immune" to storing fat and supercharged metabolism naturally. MTCH2, which has been cutely dubbed "Mitch," even ramped up muscle function and performance – described by the scientists as increased athletic ability – delivering that elusive outcome of diet-free, gym-free fat loss and improved health and fitness. Essentially, without Mitch, the muscles develop a kind of "obesity immunity," which is made even stronger thanks to the switch in metabolic function that sees cells seeking out fat to burn for energy.
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