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Hunger-blocking exercise molecule drives weight loss without workouts | And the good news is that it could be harnessed as a therapeutic, providing the same benefits without the hard work it takes to produce it naturally.
Scientists have found out exactly how an exercise-triggered molecule suppresses hunger signals in the brain, leading to weight loss. And the good news is that it could be harnessed as a therapeutic, providing the same benefits without the hard work it takes to produce it naturally.
Researchers led by scientists at Baylor College of Medicine (BCM) have unraveled just how an exercise-induced signaling metabolite, N-lactoyl-phenylalanine (Lac-Phe), travels a unique pathway to the brain and impacts the activity of one, and then two different neurons. The researchers previously found that giving added Lac-Phe to mice reduced their food intake without any negative side effects, showing that the signaling molecule still worked as if the body had produced it naturally through exercise. “This finding is important because it helps explain how a naturally produced molecule can influence appetite by interacting with a key brain region that regulates hunger and body weight,” said co-corresponding author Dr. Jonathan Long, a researcher at Stanford University School of Medicine.
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